CMA 142 Microfraction Collector

DISCONTINUED ITEM, SEE CMA 470 FRACTION COLLECTOR AS ALTERNATIVE



The CMA 142 Microfraction Collector is a unique, stand-alone instrument dedicated to microdialysis sampling.



  • Sampling from one or two probes 

  • Precision and accuracy from 1 µL 

  • Compact design 

  • Easy to use 

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DISCONTINUED ITEM, SEE CMA 470 FRACTION COLLECTOR AS ALTERNATIVE

The CMA 142 Microfraction Collector is a unique, stand-alone instrument dedicated to microdialysis sampling. Fractions ranging from 1 to 50 µL can be collected from one or two microdialysis probes (1 x 20 or 2 x 10 samples respectively). The low noise cassette movement prevents any distress to experimental animals, and the size of the instrument (130 W x 80 H x 100 D mm) permits placement close to the experiment without long connection tubing.

  • Sampling from one or two probes 
  • Precision and accuracy from 1 µL 
  • Compact design 
  • Easy to use 
Specifications
  8381143      8381142
Minimum fraction volume      1 μl 1 μl
Maximum fraction volume 50 μl 50 μl
Number of fractions 1 x 20 or 2 x 10 1 x 20 or 2 x 10
Size (W x H x D) 130 x 100 x 80 mm 130 x 100 x 80 mm
Weight 0.57 kg 0.57 kg
Power 115 V, 50 Hz 230 V, 60 Hz
Journal Articles

Recent Selected Publications

 

Gustafsson, S. et al., 2017. Combined PET and microdialysis for in vivo estimation of drug blood-brain barrier transport and brain unbound concentrations. NeuroImage, 155, pp.177–186.

 

Jin, W.-S. et al., 2017. Peritoneal dialysis reduces amyloid-beta plasma levels in humans and attenuates Alzheimer-associated phenotypes in an APP/PS1 mouse model. Acta Neuropathologica, pp.1–14.

 

Chen, X. et al., 2017. Influence of peptide transporter 2 (PEPT2) on the distribution of cefadroxil in mouse brain: A microdialysis study. Biochemical Pharmacology, 131, pp.89–97.

 

Rappeneau, V. et al., 2016. Disruption of the Glutamate–Glutamine Cycle Involving Astrocytes in an Animal Model of Depression for Males and Females. Frontiers in Behavioral Neuroscience, 10. Available at: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5147055/.

 

Rocha, J.N. & Rocha, J.N., 2016. Extracellular adenosine 5’-triphosphate concentrations changes in rat spinal cord associated with the activation of urinary bladder afferents. A microdialysis study. Einstein (São Paulo), 14(4), pp.541–546.

 

O’Leary, A. et al., 2016. Antidepressants differentially affect striatal amphetamine-stimulated dopamine and serotonin release in rats with high and low novelty-oriented behaviour. Pharmacological Research, 113, Part B, pp.739–746.

 

Justo, L.A. et al., 2016. Effects and mechanism of action of isatin, a MAO inhibitor, on in vivo striatal dopamine release. Neurochemistry International, 99, pp.147–157.

 

Brandhonneur, N. et al., 2016. PBPK model of methotrexate in cerebrospinal fluid ventricles using a combined microdialysis and MRI acquisition. European Journal of Pharmaceutics and Biopharmaceutics, 104, pp.117–130.

 

Buchanan, R.J. et al., 2016. In vivo measurements of limbic glutamate and GABA concentrations in epileptic patients during affective and cognitive tasks: A microdialysis study. Hippocampus, 26(5), pp.683–689.

 

Cremers, T.I.F.H. et al., 2016. Development of a Rat Plasma and Brain Extracellular Fluid Pharmacokinetic Model for Bupropion and Hydroxybupropion based on Microdialysis Sampling, and Application to Predict Human Brain Concentrations. Drug Metabolism and Disposition, p.dmd.115.068932.

 

Visit our Publications page for a complete listing of CMA 142 publications.

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